Sodium-bicarbonated mineral water decreases aldosterone levels without affecting urinary excretion of bone minerals

Authors: Schoppen S (1) , Pérez-Granados AM , Carbajal A , Sarriá B , Navas-Carretero S , Pilar Vaquero M
Affiliations:
(1) Department of Metabolism and Nutrition, Instituto del Frío, Spanish Council for Scientific Research (CSIC)
Source: Int J Food Sci Nutr. 2008 Jun;59(4):347-55
DOI: 10.1080/09637480701560308 Publication date: 2008 Jun E-Publication date: Not specified Availability: abstract Copyright: Not specified
Language: English Countries: Not specified Location: Not specified Correspondence address: sschoppen@fjd.es

Keywords

Article abstract

AIM: To assess in healthy postmenopausal women the influence of consuming sodium-bicarbonated mineral water on postprandial evolution of serum aldosterone and urinary electrolyte excretion.

METHODS:

Eighteen postmenopausal women consumed 500 ml of two sodium-bicarbonated mineral waters (sodium-bicarbonated mineral water 1 and sodium-bicarbonated mineral water 2) and a low-mineral water with a standard meal. Postprandial blood samples were taken at 60, 120, 240, 360 and 420 min and aldosterone concentrations were measured. Postprandial urinary minerals were determined.

RESULTS:

Urinary and total mineral excretion and urinary mineral concentrations did not differ except for sodium concentration, which was significantly higher with sodium-bicarbonated mineral water 1 than with low-mineral water (P = 0.005). There was a time effect (P = 0.003) on the aldosterone concentration. At 120 min, aldosterone concentrations were lower with sodium-bicarbonated mineral water 1 (P = 0.021) and sodium-bicarbonated mineral water 2 (P = 0.030) compared with low-mineral water.

CONCLUSION:

Drinking a sodium-rich bicarbonated mineral water with a meal increases urinary sodium concentration excretion without changes in the excretion of potassium and bone minerals.

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